发现很多童鞋对潮吹误解多多,有人说我从来木有小V高潮,可是我有潮吹的。。从科学角度来说,这个不靠谱的。潮吹来自腺体液,此腺体基本等同男性的前列腺。腺体的位置下图中会有描绘。由图可见,潮吹的前提是G点受到足够刺激,也就是G点高潮(大多数可以小V高潮的人都是通过累计G点的快感来获取Climax的,所以通常意义上的小V高潮就是G点高潮)因此,没有小V高潮就没有潮吹。很多人把小C高潮时涌出的液体当作是潮吹,事实上并非如此。能够潮吹的女人大多拥有比较强劲的盆底肌(PC),很多能够小V高潮的女人都是不能潮吹的啊,所以对于大多数的男人们来说,不要追求让你的伴侣潮吹神马的,还是先追求小V高潮吧。有人质疑G点到底存不存在,G点是存在的,科学家能扫描到这个区域。但有部分女人是没有的,比例不清楚。有些女人小V里面不管什么地方都木有快感,我的意思是说能感觉到小P在里面,也有很Full的感觉,但是木有快感,这个是因为木有找到敏感点。别担心,几乎没有女人是小V里面任何地方都有快感的,正常情况下都是有一个或者多个敏感点。要努力寻找自己小V里面的敏感点。如果真心木有敏感点,那就说明盆底肌不够强,进行Kegel锻炼会有所帮助的。发一个科普文哈,潮吹的通俗英文是Squirt,学术写法就是下文中的female ejaculation (女性射液)。文中有不少有用的信息,并且指出潮吹在防止尿道感染方面的效果。Women have glandular tissue below the bladder and surrounding the urethra that appears to be homologous to the male prostate. This tissue (also called “female prostate” or Skene’s glands) appears to the source of a viscous, white secretion, which exits from the urethra upon sexual stimulation in some women. Analysis of this secretion (also known as “female ejaculate”), and comparison with pre-coital urine from the same women, revealed that its composition was unlike urine and often contained components also found in male seminal fluid (minus the sperm). The female ejaculate had lower levels of creatinine, but had elevated levels of prostate specific antigen, prostatic acidic phosphatase, prostate specific acid phosphatase, and glucose. The functional importance of female ejaculate has yet to be fully elucidated. It is possible that retention of a prostatic tissue homolog and its glandular secretion in women is merely a vestige of development and differentiation from an embryonic, gender-neutral body plan. We hypothesize that female ejaculation has a unique function in producing a secretion into the urethra that provides protection from urinary tract infections (UTIs). We further predict that female ejaculate contains antimicrobial compounds including elements such as zinc. We also hypothesize that retention of prostatic tissue and an ability to ejaculate its glandular secretion were maintained in women because these traits provided an evolutionary advantage. Specifically: (1) women who could ejaculate antimicrobial secretions into the urethra were less likely to suffer UTIs (particularly coitus-induced UTIs), (2) women without UTIs were more likely to be receptive to coitus at a greater frequency, (3) women engaging in frequent coitus were more likely to become pregnant, and (4) women who became pregnant often were more likely to successfully reproduce the species.The existence of female ejaculation is still a matter of controversy and as of yet there has not been a clear acceptance of the existence of female ejaculation as a physiological process. The debated existence of female ejaculation is not new, but a few thousand years old. In 1672 the Dutch physician and anatomist Reinier De Graaf reported a collection of glands and ducts around the female urethra which he named the “female prostate” [1]. It was these glands that he believed produced a “pituitoserous juice” that makes “women more libidinous with its pungency and saltiness and lubricates their sexual parts in agreeable fashion during coitus.” These glands were thought to drain fluid out of the body via two pinhole-sized openings located laterally on either side of the urethra, just anterior (ventral) to the vagina.Today, most physicians know these glands by another name: Skene’s glands. De Graaf’s female prostate was renamed by the 19th century American gynecologist, Alexander Johnston Chalmers Skene [2]. In 2001, after reexamining our current state of knowledge and trying to clarify our anatomical understanding, the Federative Committee on Anatomical Terminology officially renamed the Skene’s glands, including the associated mass of tissue that surrounds the urethra, back to the original term “female prostate” (see Fig. 1). Additional support of the existence of a female prostate is evident in reports of rare cases of prostate cancer in women [3], [4], [5] and [6]. In one such case, the serum prostate specific antigen (PSA) was even elevated, 1.3 mU/ml (normal range, 0.0–0.8 mU/ml) as would be expected in a typical male prostate cancer patient [6].Fig. 1. Female pelvis sagittal with a female prostate based on sonography by Wimpissinger et al. [15].Figure optionsProponents of female ejaculation claim that the fluid produced originates from the female prostate, an organ homologous to the male prostate gland. Opponents to this view, however, dispute the existence of female ejaculation claiming instead that coital incontinence is the most likely explanation for this fluid flow. Female ejaculation and coital incontinence may only be similar superficially in perception. The two conditions are easy to confuse because the point of fluid exit is similar for both (the anterior vaginal vestibule at/near the urethral opening). Although sexual arousal/orgasm may trigger both types of fluid releases, the composition, source, and mechanism differ.Incontinence during sexual stimulation may result from increased intra-abdominal pressure (Valsalva maneuver or penile penetration) or spasms of the detrusor muscle during orgasm [7], [8] and [9]. The detrusor muscle is found within the bladder wall, and attaches to the prostate in males and the vaginal wall in females. It normally functions to help empty the bladder of urine, but may cause coital incontinence during spasm. It is unclear whether contraction of this muscle could also cause expulsion of paraurethral/prostatic glandular secretions, and therefore be responsible for female ejaculation. Interestingly, female ejaculators appear to have normal urinary continence when not sexually aroused [10] and [11].Female ejaculate has been described as a viscous fluid that is clear or milky-white, rather than the watery consistency and yellow coloration of urine [10]. Biochemical analyses indicate that female ejaculate had all of the major components of male ejaculate except sperm, including: prostate specific antigen (PSA), prostatic acidic phosphatase (PAP), prostate specific acid phosphatase (PSAP), and glucose [12], [13], [14], [15] and [16]. A lower amount of creatinine was found in female ejaculate fluid compared to urine from the same women [15] and [16].It is unclear how common the phenomenon of female ejaculation is. Estimates (based primarily on questionnaires) have reported the percentage of women who can ejaculate to be between 10% and 69% [17] and [18]. This wide range suggests confounding variables, and indicates that more research needs to be done on this topic.A sonogram of the prostate of a woman who experiences female ejaculation indicated that the shape and size of this woman’s glandular tissue is similar to that of a male prostate [15]. Hypertrophy of the paraurethral/prostatic glandular tissue may explain her ability to produce female ejaculate fluid.Many secretions produced by the human body have multiple roles. As our understanding of endogenously produced antimicrobials has grown beyond that of immunoglobulins over the last fifty years, new compounds have been found in almost all bodily fluids examined. Saliva, for example, contains a cocktail of antimicrobial agents including: lactoferrin (which helps to bind and lock away iron), lysozyme, the histatin family of compounds, and secretory IgA [19]. Other fluids, such as semen and lacrimal secretions, also incorporate a wide array of compounds that exhibit antimicrobial behaviors [19]. These also include lactoferrin and lysozyme, as well as a host of other compounds such as phospholipase A2, hCAP-18, and zinc. Zinc in particular, an especially abundant component of seminal plasma, is thought to have a broad spectrum of antimicrobial bacteriostatic and antifungal properties possibly by acting as an iron ion mimic and interfering with the normal physiological mechanisms of microbes [20], [21], [22] and [23].Whipple and Perry first suggested in 1982 the possibility that female ejaculation served an antimicrobial function Ref. [10]. Their idea may have been rooted in the same premise that rationalized the practice of voiding urine after intercourse to prevent urinary tract infections (UTIs) in women (i.e., the flow of fluids alone would wash away the microbes). Interestingly, there appears to be no scientific support for post-coital urine release as an effective measure to prevent post-coital UTIs in women [23]. However, there is documentation that coitus increases the incidence of UTIs due to the proximity of the urethral opening to the vaginal opening in the shared vaginal vestibule, combined with the mechanical effect of coital friction moving perivaginal secretions (and associated microbes) into the urethral opening [15], [16] and [23]. Once established, especially at an early age, UTIs can be hard to treat and may lead to abstention from intercourse [24].We propose that antimicrobial compounds, such as zinc that is found in seminal plasma, are similarly present in female ejaculate. The release of such antimicrobial compounds would confer a protective advantage, including a reduction in the incidence of UTIs. This has implications in human evolution, as reduction/prevention of UTIs would increase the likelihood that women would be receptive to sexual intercourse and, as a result, more likely to successfully reproduce. Thus, the maintenance of ejaculatory function in females may turn out to be much more than just a simple sexual oddity or evolutionary vestige; it may be the result of natural selection for a biological mechanism facilitating reproduction while preventing sexually transmitted microbial infections of the urinary tract.Conflicts of interest statementNone Declared.References